Long non-coding RNA emerges as a 'kill switch' for macrophages

Macrophages are considered the "scavenger cells" of the immune system, but immoderate pathogens exploit these precise cells arsenic hiding places. Researchers from the Helmholtz Institute for RNA-based Infection Research (HIRI) successful Würzburg and Philipps-Universität Marburg person discovered a mechanics that helps immune cells equilibrium their effect to infection: a agelong noncoding ribonucleic acerb called SAILR that acts arsenic an apoptosis regulator. The findings were published successful the diary PNAS.